Tham khảo Hóa sinh học arsenic

  1. ↑ Pearce, Fred (2006). When the Rivers Run Dry: Journeys Into the Heart of the World's Water Crisis. Toronto: Key Porter. ISBN 978-1-55263-741-8.
  2. 1 2 3 4 Elke Dopp, Andrew D. Kligerman & Roland A. Diaz-Bone, 2010. Organoarsenicals. Uptake, Metabolism, and Toxicity, Royal Society of Chemistry. ISBN 978-1-84973-082-2. doi:10.1039/9781849730822-00231
  3. 1 2 Wilcox, Dean E. (2013). “Chapter 15. Arsenic. Can This Toxic Metalloid Sustain Life?”. Trong Astrid Sigel, Helmut Sigel & Roland K. O. Sigel (biên tập). Interrelations between Essential Metal Ions and Human Diseases. Metal Ions in Life Sciences. 13. Springer. tr. 475–498. doi:10.1007/978-94-007-7500-8_15.
  4. ↑ Arsenocholine - Cấu trúc và Dữ liệu
  5. ↑ Francesconi, Kevin A.; Edmonds, John S.; Stick, Robert V. (1992). “Arsenic compounds from the kidney of the giant clam Tridacna maxima: Isolation and identification of an arsenic-containing nucleoside”. Journal of the Chemical Society, Perkin Transactions 1 (11): 1349. doi:10.1039/P19920001349.
  6. ↑ Bentley, Ronald; Chasteen, TG (2002). “Microbial Methylation of Metalloids: Arsenic, Antimony, and Bismuth”. Microbiology and Molecular Biology Reviews. 66 (2): 250–271. doi:10.1128/MMBR.66.2.250-271.2002. PMC 120786. PMID 12040126.
  7. ↑ Cullen, William R; Reimer, Kenneth J. (1989). “Arsenic speciation in the environment”. Chemical Reviews. 89 (4): 713–764. doi:10.1021/cr00094a002.
  8. ↑ Ronald Bentley & Thomas G. Chasteen (2002). “Microbial Methylation of Metalloids: Arsenic, Antimony, and Bismuth”. Microbiology and Molecular Biology Reviews. 66 (2): 250–271. doi:10.1128/MMBR.66.2.250-271.2002. PMC 120786. PMID 12040126.
  9. ↑ Cullen, William R.; Reimer, Kenneth J. (1989). “Arsenic speciation in the environment”. Chemical Reviews. 89: 713–764. doi:10.1021/cr00094a002.
  10. ↑ Oremland, Ronald S.; Saltikov, Chad W.; Wolfe-Simon, Felisa; Stolz, John F. (2009). “Arsenic in the Evolution of Earth and Extraterrestrial Ecosystems”. Geomicrobiology Journal. 26 (7): 522–536. doi:10.1080/01490450903102525.
  11. ↑ Erb, T. J.; Kiefer, P.; Hattendorf, B.; Gunther, D.; Vorholt, J. A. (2012). “GFAJ-1 is an Arsenate-Resistant, Phosphate-Dependent Organism”. Science. 337 (6093): 467–70. Bibcode:2012Sci...337..467E. doi:10.1126/science.1218455. PMID 22773139.
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  13. ↑ Westheimer, F.H. (ngày 6 tháng 6 năm 1987). “Why nature chose phosphates” (PDF). Science. 235 (4793): 1173–1178 (see pp. 1175–1176). Bibcode:1987Sci...235.1173W. doi:10.1126/science.2434996. Bản gốc (PDF) lưu trữ ngày 16 tháng 6 năm 2011.
  14. ↑ Nordstrom D. K. (2002). “Worldwide occurrences of arsenic in ground water”. Science. 296 (5576): 2143–2145. doi:10.1126/science.1072375.
  15. ↑ Hileman, B (ngày 9 tháng 4 năm 2007). “Arsenic in Chicken Production”. Chemical and Engineering News. tr. 34–35.
  16. ↑ Bottemiller, Helena (ngày 26 tháng 9 năm 2009). “Bill Introduced to Ban Arsenic Antibiotics in Feed”. Food Safety News. Truy cập ngày 10 tháng 1 năm 2011.
  17. 1 2 Sakurai T (2003). “Biomethylation of Arsenic is Essentially Detoxicating Event”. Journal of Health Science. 49 (3): 171–178. doi:10.1248/jhs.49.171. Truy cập ngày 10 tháng 1 năm 2011.
  18. ↑ Jun Zhu; Zhu Chen; Valérie Lallemand-Breitenbach; Hugues de Thé (2002). “How Acute Promyelocytic Leukaemia Revived Arsenic”. Nature Reviews Cancer. 2 (9): 705–714. doi:10.1038/nrc887. PMID 12209159. Bản gốc lưu trữ ngày 19 tháng 10 năm 2017. Truy cập ngày 27 tháng 9 năm 2013.
  19. 1 2 Gibaud, Stéphane; Jaouen, Gérard (2010). “Arsenic - based drugs: from Fowler's solution to modern anticancer chemotherapy”. Topics in Organometallic Chemistry. Topics in Organometallic Chemistry. 32: 1–20. doi:10.1007/978-3-642-13185-1_1. ISBN 978-3-642-13184-4.
  20. ↑ Elschenbroich C., 2006. "Organometallics". Wiley-VCH: Weinheim. ISBN 978-3-527-29390-2
  21. 1 2 Park, Woo H. Park; Jae G. Seol; Eun S. Kim; Jung M. Hyun; Chul W. Jung; Chung C. Lee; Byoung K. Kim; Young Y. Lee (ngày 6 tháng 6 năm 2000). “Arsenic Trioxide-mediated Growth Inhibition in MC/CAR Myeloma Cells via Cell Cycle Arrest in Association with Induction of Cyclin-dependent Kinase Inhibitor, p21, and Apoptosis”. Cancer Research. 60 (3065): 3065–71. PMID 10850458. Truy cập ngày 15 tháng 12 năm 2010.
  22. ↑ Lunghi, Paolo Lunghi; Antonio Costanzo; Massimo Levrero; Antonio Bonati (ngày 15 tháng 7 năm 2004). “Treatment with arsenic trioxide (ATO) and MEK1 inhibitor activates the p73-p53AIP1 apoptotic pathway in leukemia cells”. Blood. 104 (2): 519–525. doi:10.1182/blood-2003-08-2743. PMID 15031205. Truy cập ngày 15 tháng 12 năm 2010.
  23. ↑ “Arsenic in Drinking Water - Review article” (PDF). IARC Monographs - World Health Organization. 84: 133–135. Truy cập ngày 10 tháng 1 năm 2011.
  24. 1 2 3 “Arsenic in Drinking Water - Review article” (PDF). IARC Monographs - World Health Organization. 84. Truy cập ngày 10 tháng 1 năm 2011.
  25. 1 2 3 4 5 6 7 8 9 10 Kumagai, Yoshito; Sumi, Daigo (2007). “Arsenic: Signal Transduction, Transcription Factor, and Biotransformation Involved in Cellular Response and Toxicity”. Annual Review of Pharmacology and Toxicology. 47: 243–62. doi:10.1146/annurev.pharmtox.47.120505.105144. PMID 17002598.
  26. 1 2 Vahter, Marie (2002). “Mechanisms of arsenic biotransformation”. Toxicology. 181–182: 211–7. doi:10.1016/S0300-483X(02)00285-8. PMID 12505313.
  27. 1 2 3 4 5 6 7 8 9 10 11 12 13 Hunt, Katherine M.; Srivastava, Ritesh K.; Elmets, Craig A.; Athar, Mohammad (2014). “The mechanistic basis of arsenicosis: Pathogenesis of skin cancer”. Cancer Letters. 354 (2): 211–9. doi:10.1016/j.canlet.2014.08.016. PMID 25173797.
  28. ↑ Petrick, Jay S.; Ayala-Fierro, Felix; Cullen, William R.; Carter, Dean E.; Vasken Aposhian, H. (2000). “Monomethylarsonous Acid (MMAIII) is More Toxic Than Arsenite in Chang Human Hepatocytes”. Toxicology and Applied Pharmacology. 163 (2): 203–7. doi:10.1006/taap.1999.8872. PMID 10698679.
  29. ↑ “Arsenic in Drinking Water - Review article” (PDF). IARC Monographs - World Health Organization. 84: 138. Truy cập ngày 10 tháng 1 năm 2011.
  30. ↑ Ajees, A.A.; và đồng nghiệp (ngày 10 tháng 7 năm 2012). “Structure of an As(III) S-Adenosylmethionine Methyltransferase: insights into the Mechanism of Arsenic Biotransformation”. Biochemistry. 51: 5476–5485. doi:10.1021/bi3004632.
  31. ↑ Kumagai, Yoshito; Sumi, Daigo Sumi (2007). “Arsenic: Signal Transduction, Transcription Factor, and Biotransformation Involved in Cellular Response and Toxicity”. Annual Review of Pharmacology and Toxicology. 47: 243–62. doi:10.1146/annurev.pharmtox.47.120505.105144. PMID 17002598.
  32. ↑ Itoh, K.; Wakabayashi, N.; Katoh, Y.; Ishii, T.; Igarashi, K.; Engel, J. D.; Yamamoto, M (1999). “Keap1 represses nuclear activation of antioxidant responsive elements by Nrf2 through binding to the amino-terminal Neh2 domain”. Genes Dev. 13 (1): 76–86. doi:10.1101/gad.13.1.76. PMC 316370. PMID 9887101.
  33. 1 2 Kumagai, Yoshito; Sumi, Daigo Sumi (2007). “Arsenic: Signal Transduction, Transcription Factor, and Biotransformation Involved in Cellular Response and Toxicity”. Annual Review of Pharmacology and Toxicology. 47: 243–62. doi:10.1146/annurev.pharmtox.47.120505.105144. PMID 17002598.
  34. ↑ Pi, J; Waalkes, MP; Kumagai, Y; Reece, JM; Qu, W (2003). “Transcription factor Nrf2 activation by inorganic arsenic in cultured keratinocytes: involvement of hydrogen peroxide”. Exp. Cell Res. 290 (2): 234–45. doi:10.1016/s0014-4827(03)00341-0. PMID 14567983.
  35. ↑ Stýblo, M.; Drobná, Z.; Jaspers, I.; Lin, S.; Thomas, D. J. (2002). “The role of biomethylation in toxicity and carcinogenicity of arsenic: A research update”. Environmental Health Perspectives. 110 (Suppl 5): 767–771. doi:10.1289/ehp2110s5767. PMC 1241242. PMID 12426129.
  36. ↑ Chouchane, S.; Snow, E. T.; Snow, E. T. “In vitro effect of arsenical compounds on glutathione-related enzymes”. Chem. Res. Toxicol. 14 (5): 517–22. PMID 11368549.
  37. ↑ Petrick, Jay S.; Jagadish, Bhumasamudram; Mash, Eugene A.; Aposhian, H. Vasken (2001). “Monomethylarsonous Acid (MMAIII) and Arsenite:  LD50in Hamsters and in Vitro Inhibition of Pyruvate Dehydrogenase”. Chemical Research in Toxicology. 14 (6): 651–656. doi:10.1021/tx000264z. PMID 11409934.
  38. ↑ Lin, Lin S.; Thomas, D. J.; Cullen, W. R.; Wang, C.; Styblo, M.; Del Razo, L. M. (2001). “Arsenicals inhibit thioredoxin reductase in cultured rat hepatocytes”. Chem. Res. Toxicol. 14 (3): 305–11. doi:10.1021/tx0001878. PMID 11258980.
  39. ↑ Cullen, William R; Reimer, Kenneth J. (1989). “Arsenic speciation in the environment”. Chemical Reviews. 89 (4): 713–764. doi:10.1021/cr00094a002.
  40. 1 2 3 4 5 6 7 8 9 10 11 12 13 14 Hughes, Michael F (2002). “Arsenic toxicity and potential mechanisms of action”. Toxicology Letters. 133 (1): 1–16. doi:10.1016/S0378-4274(02)00084-X. PMID 12076506.
  41. ↑ Rossman, T.G (2003). “Mechanism of arsenic carcinogenesis: An integrated approach”. Mutation Research/Fundamental and Molecular Mechanisms of Mutagenesis. 533: 37–65. doi:10.1016/j.mrfmmm.2003.07.009.
  42. ↑ Pierce, B.L; Kibriya, M.G (2012). “Genome-wide association study identifies chromosome 10q24.32 variants associated with arsenic metabolism and toxicity phenotypes in Bangladesh”. PLoS Genetics. 8: e1002522. doi:10.1371/journal.pgen.1002522.
  43. ↑ Li, J.H; Rossman, T.G (1991). “Comutagenesis of sodium arsenite with ultraviolet radiation in Chinese hamster V79 cells”. Biology of Metals. 4: 197–200. doi:10.1007/BF01141180.
  44. ↑ Lee, T.C; Oshimura, M (1985). “Comparison of arsenic-induced cell transformation, cytotoxicity, mutation and cytogenetic effects in Syrian hamster embryo cells in culture”. Carcinogenesis. 6: 1421–1426. doi:10.1093/carcin/6.10.1421.
  45. ↑ Kessel, M; Liu, S.X (2002). “Arsenic induces oxidative DNA damage in mammalian cells”. Molecular and Cellular Biochemistry. 234/235: 234–235:301–308. doi:10.1023/A:1015927406142.
  46. ↑ Nesnow, S; Roop, B.C (2002). “DNA damage induced by methylated trivalent arsenicals is mediated by reactive oxygen species”. Chemical Research in Toxicology. 15: 1627–1634. doi:10.1021/tx025598y.
  47. ↑ Jomova, K; Jenisova, Z (2011). “Arsenic: Toxicity, oxidative stress and human disease”. Journal of Applied Toxicology. 31: 95–107. doi:10.1002/jat.1649.
  48. ↑ Kitchin, K.T; Wallace, K (2008). “Evidence against the nuclear in situ binding of arsenicals—Oxidative stress theory of arsenic carcinogenesis”. 232: 252–257. doi:10.1016/j.taap.2008.06.02. Chú thích journal cần |journal= (trợ giúp)
  49. ↑ Bau, D.T; Wang, T.S (2002). “Oxidative DNA adducts and DNA-protein cross-links are the major DNA lesions induced by arsenite”. Environmental Health Perspectives. 110: 753–756. doi:10.1289/ehp.02110s5753. PMC 1241239. PMID 12426126.
  50. ↑ Hwang, E.S; Kim, G.H (2007). “Biomarkers for oxidative stress status of DNA, lipids, and proteins in vitro and in vivo cancer research”. Toxicology. 229: 1–10. doi:10.1016/j.tox.2006.10.013.
  51. ↑ Liu, Su X. (tháng 12 năm 2000). “Induction of oxyradicals by arsenic: Implication for mechanism of genotoxicity”. Proceedings of the National Academy of Sciences of the United States of America. Truy cập ngày 4 tháng 4 năm 2013.
  52. ↑ Grollman, A.P; Moriya, M (1993). “Mutagenesis by 8-oxoguanine: An enemy within”. Trends in Genetics. 9: 246–249. doi:10.1016/0168-9525(93)90089-Z.
  53. ↑ Martinez, V.D; Vucic, E.A (2011). “Arsenic biotransformation as a cancer promoting factor by inducing DNA damage and disruption of repair mechanisms”. Molecular Biology International. 2011: 1–11. doi:10.4061/2011/718974.
  54. ↑ Lai, Y; Zhao, W (2011). “Role of DNA polymerase beta in the genotoxicity of arsenic”. Environmental and Molecular Mutagenesis. 52: 460–468. doi:10.1002/em.20643.
  55. ↑ Hartwig, A; Groblinghoff, U.D (1997). “Interaction of arsenic(III) with nucleotide excision repair in UV-irradiated human fibroblasts”. Carcinogenesis. 18: 399–405. doi:10.1093/carcin/18.2.399. PMID 9054635.
  56. ↑ Curnow, A; Salter, L (2001). “A preliminary investigation of the effects of arsenate on irradiation-induced DNA damage in cultured human lung fibroblasts”. Journal of Toxicology and Environmental Health, Part A. 63: 605–616. doi:10.1080/152873901316857789.
  57. ↑ Schwerdtle, T; Walter, I (2003). “Induction of oxidative DNA damage by arsenite and its trivalent and pentavalent methylated metabolites in cultured human cells and isolated DNA”. Carcinogenesis. 24: 967–974. doi:10.1093/carcin/bgg018.
  58. ↑ Lai, Y; Zhao, W (2011). “Role of DNA polymerase beta in the genotoxicity of arsenic”. Environmental and Molecular Mutagenesis. 52: 460–468. doi:10.1002/em.20643.
  59. 1 2 Ebert, F; Weiss, A (2011). “Arsenicals affect base excision repair by several mechanisms”. Mutat Res. 715: 32–41. doi:10.1016/j.mrfmmm.2011.07.004. PMID 21782832.
  60. ↑ Sykora, P; Snow, E.T (2008). “Modulation of DNA polymerase beta-dependent base excision repair in cultured human cells after low dose exposure to arsenite”. Toxicology and Applied Pharmacology. 228: 385–394. doi:10.1016/j.taap.2007.12.019.
  61. 1 2 3 4 5 6 Hunt, K. M; Srivastava, R. K; Elmets, C. A; Athar, M. (2014). “The mechanistic basis of arsenicosis: Pathogenesis of skin cancer”. Cancer Letters. 354: 211–219. doi:10.1016/j.canlet.2014.08.016. PMID 25173797.
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  64. ↑ “Acute & Chronic Poisoning Affects”. medtox. Truy cập ngày 30 tháng 3 năm 2015.

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WikiPedia: Hóa sinh học arsenic http://www.foodsafetynews.com/2009/09/bill-introdu... http://www.nature.com/nrc/journal/v2/n9/full/nrc88... http://link.springer.com/chapter/10.1007%2F978-3-6... http://academic.evergreen.edu/curricular/m2o2006/s... http://monographs.iarc.fr/ENG/Monographs/vol84/mon... //pubmed.ncbi.nlm.nih.gov/10698679 //pubmed.ncbi.nlm.nih.gov/10850458 //pubmed.ncbi.nlm.nih.gov/11258980 //pubmed.ncbi.nlm.nih.gov/11368549 //pubmed.ncbi.nlm.nih.gov/11409934